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Shoulder Examination in Rheumatology

📋 Key Information Summary

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  • The shoulder comprises four articulations — glenohumeral (GH), acromioclavicular (AC), sternoclavicular (SC), and scapulothoracic — all of which must be systematically assessed.
  • Rotator cuff (SITS: supraspinatus, infraspinatus, teres minor, subscapularis) is the most common source of shoulder pathology in rheumatology practice.
  • Use the Look–Feel–Move approach: inspect for asymmetry, swelling, and atrophy; palpate bony and soft-tissue landmarks; assess active then passive ROM.
  • Normal glenohumeral ROM: flexion 180°, abduction 180°, external rotation 60–90°, internal rotation 60–90° (to T6–T12 spinous processes).
  • Always compare active and passive ROM — preserved passive ROM with limited active ROM suggests a tendon/muscle pathology rather than joint contracture.
  • The painful arc (pain between 60° and 120° of abduction) is a hallmark of subacromial pathology including rotator cuff tendinopathy and bursitis.
  • Frozen shoulder (adhesive capsulitis) presents with global restriction of BOTH active and passive ROM in all planes — this distinguishes it from rotator cuff disease.
  • Inflammatory shoulder pathology typically causes bilateral symptoms, morning stiffness >30 minutes, and systemic features — consider RA, PMR, and seronegative spondyloarthropathy.
  • Key special tests: Neer and Hawkins-Kennedy (impingement), empty can/Jobe (supraspinatus), external rotation lag sign (infraspinatus), internal rotation lag sign (subscapularis), Speed's test (long head of biceps), O'Brien (SLAP / AC joint).
  • The Constant–Murley score provides a validated patient-reported and clinician-assessed shoulder function score (0–100 points).
  • Ultrasound is the first-line imaging for rotator cuff pathology; plain radiographs (AP, axillary lateral) assess bony pathology, calcific tendinitis, and OA.
  • Aboriginal and Torres Strait Islander peoples experience higher rates of shoulder musculoskeletal disease with reduced access to specialist rheumatology and imaging services.

Introduction & Australian Epidemiology

A structured shoulder examination is essential for differentiating inflammatory, mechanical, and periarticular causes of shoulder pain in clinical rheumatology and musculoskeletal medicine practice. The shoulder is the third most common site of musculoskeletal consultation in Australian general practice, accounting for approximately 11% of all musculoskeletal presentations.

Shoulder complaints are responsible for significant functional impairment, lost work productivity, and reduced quality of life. In Australia, rotator cuff disease affects an estimated 20–30% of the adult population, with prevalence increasing with age — reaching 50–60% in those over 60 years. Frozen shoulder (adhesive capsulitis) has an estimated prevalence of 2–5%, with higher rates in people with diabetes mellitus, thyroid disease, and prolonged immobilisation.

The anatomical complexity of the shoulder — involving four discrete articulations and multiple periarticular structures — demands a systematic, structured examination approach. In rheumatology practice, distinguishing inflammatory arthropathy from degenerative or periarticular pathology is critical, as the investigation and management pathways diverge significantly. This guideline provides a practical framework for the clinical examination of the shoulder, with emphasis on Australian practice patterns, imaging availability, and validated scoring systems.

Shoulder Examination in Rheumatology clinical infographic — pathophysiology, clinical clues, diagnosis, imaging, and management
Tap or click image to enlarge — Shoulder Examination in Rheumatology: pathophysiology, clinical clues, diagnosis, imaging, and management.
Shoulder Examination in Rheumatology infographic, full size

Anatomy & Functional Assessment

Articulations

The shoulder complex comprises four distinct articulations, each of which contributes to the overall range of motion and must be individually assessed during clinical examination:

Joint Type Key Features Examination Significance
Glenohumeral (GH) Ball-and-socket synovial Glenoid labrum provides socket depth; greatest ROM of any joint Primary joint for ROM testing; intra-articular pathology (RA, OA, frozen shoulder)
Acromioclavicular (AC) Plane synovial Stabilised by AC ligament and coracoclavicular ligaments Cross-body adduction test localises AC joint pathology
Sternoclavicular (SC) Saddle synovial Only axial articulation with the trunk; articular disc Swelling may indicate septic arthritis, RA, or SAPHO syndrome
Scapulothoracic Functional (non-synovial) Serratus anterior, trapezius, rhomboids, levator scapulae stabilise Winged scapula (long thoracic nerve palsy); scapular dyskinesis

Rotator Cuff — SITS Muscles

The rotator cuff muscles form a musculotendinous cuff around the GH joint, providing dynamic stabilisation and enabling glenohumeral movement. The mnemonic SITS describes the four muscles:

Muscle Nerve Primary Action Clinical Test
Supraspinatus Suprascapular (C5–C6) Initiates abduction (first 15°) Empty can / Jobe test
Infraspinatus Suprascapular (C5–C6) External rotation External rotation lag sign
Teres minor Axillary (C5–C6) External rotation, adduction Hornblower's sign
Subscapularis Upper & lower subscapular (C5–C7) Internal rotation Internal rotation lag sign; Belly-press test; Lift-off test

The Look–Feel–Move Approach

A systematic shoulder examination follows the structured Look–Feel–Move (Inspection–Palpation–Movement) framework, supplemented by special tests specific to the suspected pathology.

1
Look (Inspection)
  • Expose both shoulders; stand behind the patient
  • Assess symmetry — compare side-to-side
  • Muscle wasting: supraspinatus/infraspinatus atrophy (suprascapular neuropathy, massive rotator cuff tear), deltoid atrophy (axillary nerve palsy)
  • Scapular winging (long thoracic nerve — serratus anterior)
  • Visible swelling — anterior (GH joint effusion), superior (AC joint), SC joint
  • Skin changes: erythema (septic arthritis, gout), psoriatic plaques, surgical scars
  • Posture: rounded shoulders, forward head carriage
2
Feel (Palpation)
  • AC joint — palpate along the clavicle to the acromion; tenderness localises AC pathology
  • Bicipital groove — palpate in the bicipital groove (between greater and lesser tuberosities) anteriorly; tenderness suggests biceps tendinopathy
  • Greater tuberosity — lateral aspect; tenderness over supraspinatus insertion
  • Subacromial space — palpate through the deltoid; tenderness suggests bursitis
  • GH joint line — palpate anteriorly through the rotator interval
  • Suprascapular notch — posterior triangle; tenderness may indicate suprascapular nerve entrapment
  • Temperature: increased warmth over inflamed joint (RA, septic arthritis)
3
Move (Range of Motion)
  • Always assess ACTIVE ROM first, then passive ROM — the discrepancy is diagnostic
  • Compare with the contralateral (unaffected) shoulder
  • Document range using a goniometer where possible
  • Assess rhythm: scapulohumeral rhythm is normally 2:1 (GH : scapulothoracic contribution)

Normal Glenohumeral Range of Motion

Movement Normal ROM Muscles Involved Common Restriction Causes
Flexion 180° Anterior deltoid, supraspinatus Frozen shoulder, GH arthritis, rotator cuff disease
Abduction 180° Deltoid, supraspinatus (initiates first 15°) Supraspinatus tear, impingement, frozen shoulder
External rotation 60–90° Infraspinatus, teres minor Frozen shoulder (early loss), subscapularis tear
Internal rotation 60–90° Subscapularis, pectoralis major, latissimus dorsi Infraspinatus/supraspinatus pathology, adhesive capsulitis
Internal rotation (spinous process level) T6–T12 Subscapularis, teres major Frozen shoulder, GH arthritis
Extension 40–60° Posterior deltoid, latissimus dorsi Frozen shoulder, posterior capsule tightness

Active vs Passive ROM — Diagnostic Significance

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Key diagnostic principle: The relationship between active and passive ROM is the single most important differentiator in shoulder examination:
  • Active = Passive (both restricted) → Joint pathology (frozen shoulder, GH arthritis, capsular contracture)
  • Active < Passive (pain/weakness limiting active) → Muscle/tendon pathology (rotator cuff tendinopathy or tear)
  • Active = Passive (both full) with pain → Bursitis, early impingement, or referred pain

The Painful Arc

The painful arc test is performed by asking the patient to actively abduct the arm from 0° to 180°. Pain occurring specifically between 60° and 120° of abduction is characteristic of subacromial pathology (rotator cuff tendinopathy, subacromial bursitis, or calcific tendinitis). Pain at the extremes of ROM (beginning and end range) more commonly suggests capsular pathology.

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Clinical pearl: The painful arc is a screening test with high sensitivity (~70–80%) but low specificity. A positive painful arc should prompt targeted rotator cuff testing and does not confirm a specific diagnosis in isolation. Always combine with resisted strength testing and palpation.

Special Tests

Special tests are provocative manoeuvres designed to stress specific anatomical structures to reproduce symptoms and localise pathology. Each test should be interpreted in the context of the overall clinical picture, not in isolation. The table below summarises the most clinically useful shoulder special tests in rheumatology practice.

Subacromial Impingement Tests

Test Technique Positive Finding Structures Assessed Test Performance
Neer's Impingement Stabilise scapula with one hand; passively flex the arm maximally with the other Pain at maximal flexion Supraspinatus tendon against coracoacromial arch Sensitivity 75–89%, Specificity 31–48%
Hawkins–Kennedy Arm at 90° forward flexion; passively internally rotate the shoulder Pain on internal rotation Rotator cuff tendons / bursa under coracoacromial ligament Sensitivity 72–92%, Specificity 25–66%
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Clinical use: Combining Neer and Hawkins–Kennedy — if both are positive, the post-test probability of impingement increases significantly. If both are negative, subacromial impingement is unlikely (high negative predictive value).

Rotator Cuff Integrity Tests

Test Technique Positive Finding Structures Assessed Test Performance
Empty Can / Jobe Test Arm at 90° abduction, 30° forward flexion, thumb pointing down (pronated); resist downward pressure Pain and/or weakness against resistance Supraspinatus tendon Sensitivity 41–91%, Specificity 50–90%
External Rotation Lag Sign Passively position arm at 20° abduction with 90° external rotation; ask patient to hold; release Arm drops/rotates internally (loss of ER position) Infraspinatus / teres minor (massive tear) Specificity ~90%+ for full-thickness infraspinatus tear
Internal Rotation Lag Sign Passively position arm at maximum internal rotation (hand behind back); ask patient to hold; release Hand drops / cannot maintain position Subscapularis Specificity ~90%+ for subscapularis tear
Belly-Press Test Patient presses hand firmly against abdomen with elbow anterior to the trunk Unable to maintain pressure; elbow drops posteriorly or compensatory wrist flexion Subscapularis (upper portion) Sensitivity 40–80%, Specificity 60–90%
Lift-Off Test (Gerber) Hand placed behind the back (lumbar region); ask patient to lift hand off back Unable to lift hand away from back Subscapularis (lower portion) Specificity ~85%+ but cannot be performed if ROM restricted
Hornblower's Sign Arm at 90° abduction, elbow at 90° flexion; ask patient to externally rotate against resistance Unable to maintain external rotation position Teres minor Specificity ~93% for teres minor pathology
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Lag signs indicate full-thickness tears: A positive external or internal rotation lag sign has high specificity (>90%) for full-thickness rotator cuff tears and should prompt urgent ultrasound or MRI referral. These signs are more reliable than resisted strength tests alone for identifying complete tendon rupture.

Biceps Tendon Tests

Test Technique Positive Finding Structures Assessed
Speed's Test Arm at 90° forward flexion, elbow extended, supinated forearm; resist downward force Pain in bicipital groove Long head of biceps (bicipital tendinopathy)
Yergason's Test Elbow flexed to 90°, forearm pronated; resist supination while stabilising bicipital groove Pain and/or subluxation in bicipital groove Biceps tendon / transverse humeral ligament

Instability Tests

Test Technique Positive Finding Structures Assessed
Apprehension Test Arm at 90° abduction; slowly externally rotate Patient becomes apprehensive / resists further ER; sense of impending dislocation Anterior GH instability
Relocation Test Follows apprehension test: apply posterior force to humeral head while at maximal ER Apprehension resolves; further ER possible Confirms anterior instability (Bankart lesion)

AC Joint and Labral Tests

Test Technique Positive Finding Structures Assessed
O'Brien's Test (Active Compression) Arm at 90° flexion, 10–15° adduction; first with thumb down (pronated), then thumb up (supinated); resist downward force in both positions Pain with thumb down > thumb up; pain localised superiorly = AC joint; pain/deep click = labral (SLAP) AC joint pathology / SLAP lesion (superior labral tear)
Cross-Body Adduction Passively adduct the arm across the body at 90° flexion Pain localised to AC joint AC joint OA, distal clavicle osteolysis

Scapular Assessment

Scapular dyskinesis (abnormal scapular motion) contributes to shoulder pathology and should be assessed during active arm elevation:

  • Observe scapulohumeral rhythm from behind during arm elevation — should be 2:1 ratio
  • Lateral scapular slide test: measure distance from inferior angle to spinous process at 0°, 45° abduction, and 90° abduction; asymmetry >1.5 cm suggests scapular dyskinesis
  • Winged scapula: ask patient to push against a wall; medial winging = long thoracic nerve palsy (serratus anterior); lateral winging = spinal accessory nerve palsy (trapezius)

Clinical Patterns — Diagnostic Algorithms

The constellation of examination findings — rather than any single test — determines the clinical diagnosis. The following clinical patterns represent the most common diagnostic scenarios encountered in rheumatology practice.

Pattern Recognition Table

Clinical Pattern Active ROM Passive ROM Pain Pattern Strength Key Distinguishing Features
Frozen Shoulder ↓↓ Globally restricted ↓↓ Globally restricted Diffuse, aching; night pain Cannot assess (limited by ROM) Loss of external rotation is the hallmark; capsular pattern (ER > abduction > IR); 3 phases (painful → stiff → thawing)
Rotator Cuff Tendinopathy / Tear ↓ Painful arc Normal or near-normal 60–120° abduction (painful arc) ↓ Resisted weakness (muscle-specific) Active < Passive = tendon pathology; positive Jobe / ER lag / IR lag
Subacromial Bursitis ↓ Painful arc Near normal 60–120° abduction; pain on IR in abduction Normal (no weakness) Painful arc + no weakness = bursitis; resisted tests may be painful but not weak
Inflammatory Arthropathy (RA, PMR) ↓ Bilateral ↓ Bilateral Bilateral, diffuse; morning stiffness >30 min May be reduced Bilateral + morning stiffness + systemic features (fatigue, weight loss); synovial thickening on palpation; consider PMR if age >50 with bilateral shoulder girdle pain
Calcific Tendinitis ↓ Acute pain ↓ (pain limits) Acute, severe; night pain; may mimic septic arthritis Cannot test (pain) Sudden acute onset; radiographic calcification in supraspinatus; self-limiting (resorptive phase)
GH Osteoarthritis ↓ Gradual Deep, aching; worse with activity May be reduced Age >60; crepitus; capsular pattern; X-ray shows joint space narrowing, osteophytes, sclerosis

Frozen Shoulder — The Hallmark of Global Restriction

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Key diagnostic criterion: Frozen shoulder is the only common condition that restricts BOTH active AND passive ROM equally in all planes. The hallmark finding is loss of external rotation — if a patient cannot externally rotate the arm at all, frozen shoulder should be the working diagnosis until proven otherwise. This distinguishes it from rotator cuff pathology, where passive ROM is preserved.

Three phases of frozen shoulder:

1
Phase 1 — Freezing (Painful) Phase
Duration: 2–9 months. Progressive onset of diffuse shoulder pain with gradual loss of ROM. Pain is present at rest and often disrupts sleep. Patient is troubled by pain more than stiffness.
2
Phase 2 — Frozen (Stiff) Phase
Duration: 4–12 months. Pain diminishes but stiffness dominates. Global loss of ROM (especially ER and IR). Functional limitation is severe — difficulty with overhead activities, reaching behind back, and dressing.
3
Phase 3 — Thawing (Recovery) Phase
Duration: 5–24 months. Gradual return of ROM. May take 1–3 years for full resolution. Most patients achieve functional ROM even without treatment. Permanent stiffness occurs in ~10%.

Rotator Cuff Pathology — The Painful Arc + Weakness Pattern

Rotator cuff disease represents a spectrum from tendinopathy to partial-thickness tears to full-thickness tears. The examination pattern is:

  • Painful arc (60–120° abduction) — present in tendinopathy and tears
  • Resisted weakness — specific to the involved tendon (Jobe for supraspinatus, ER for infraspinatus, IR for subscapularis)
  • Passive ROM preserved — this is the critical differentiator from frozen shoulder
  • Lag signs — indicate full-thickness tears (high specificity)

Bursitis vs Tendinopathy

Subacromial bursitis and rotator cuff tendinopathy frequently coexist but can be distinguished clinically:

  • Bursitis alone: Painful arc, pain on Neer/Hawkins, but no weakness on resisted testing
  • Tendinopathy: Painful arc plus weakness on resisted testing of the specific tendon
  • Bursitis: Pain with passive internal rotation of the abducted arm (Hawkins–Kennedy positive)
  • Both improve with subacromial injection of local anaesthetic ± corticosteroid (diagnostic injection)

Inflammatory Shoulder — Red Flags for Systemic Disease

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Consider inflammatory aetiology when:
  • Bilateral shoulder pain and stiffness
  • Morning stiffness >30 minutes
  • Systemic features: fatigue, weight loss, low-grade fever, night sweats
  • Joint swelling / synovial thickening on palpation
  • Age >50 with bilateral shoulder girdle + hip girdle pain → consider polymyalgia rheumatica (PMR)
  • Symmetrical small joint involvement → consider rheumatoid arthritis
  • Order: FBC, ESR, CRP, RF, anti-CCP antibodies, ANA; consider ultrasound for subdeltoid bursitis / GH synovitis

Constant–Murley Shoulder Score

The Constant–Murley score is the most widely used validated outcome measure for shoulder function. It provides a composite score (0–100 points) incorporating subjective and objective components:

Domain Components Max Score Assessment Method
Pain Severity of pain (subjective) 15 points Visual analogue scale (0 = severe pain, 15 = no pain)
Activities of Daily Living Work, recreation, sleep position 20 points Patient-reported questionnaire
Range of Motion Flexion, abduction, ER, IR 40 points Clinician-assessed with goniometer
Strength Isometric abduction strength 25 points Measured with spring balance or dynamometer at 90° abduction

Interpretation: 90–100 = excellent; 80–89 = good; 70–79 = fair; <70 = poor. Age- and sex-adjusted normal values are available. The Constant–Murley score is used for monitoring treatment outcomes in rotator cuff repair, arthroplasty, and frozen shoulder management. It is less useful in acute settings where pain limits full testing.

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Australian practice note: The Constant–Murley score is commonly used in Australian orthopaedic and rheumatology practice for pre- and post-operative assessment. It is recommended by the Australian Orthopaedic Association (AOA) shoulder arthroplasty registry. Ensure the isometric strength component is measured with a calibrated dynamometer for reproducible results.

Investigations & Imaging

Clinical examination guides investigation selection. Imaging should be directed by the working diagnosis derived from examination findings, not used as a screening tool for all shoulder pain.

Laboratory Investigations

MBS Available
Full Blood Count (FBC)
Leucocytosis (septic arthritis), anaemia of chronic disease (RA, malignancy). MBS Item 65070.
MBS Available
ESR and CRP
Elevated in inflammatory conditions (RA, PMR, septic arthritis). ESR >40 + bilateral shoulder pain → consider PMR. MBS Item 65070.
MBS Available
Rheumatoid Factor (RF) & Anti-CCP Antibodies
Suspected RA. Anti-CCP more specific than RF for RA diagnosis. MBS Item 65070.
MBS Available
Uric Acid
Suspected gouty arthropathy (monoarticular, acute, male). MBS Item 65070.
MBS Available
HbA1c / Fasting Glucose
Screen for diabetes — major risk factor for frozen shoulder. MBS Item 65070.
MBS Available
TSH
Thyroid disease is associated with frozen shoulder. MBS Item 65070.

Imaging

MBS Available
Plain Radiographs — AP and Axillary Lateral
First-line for bony pathology. Assess: GH joint space (OA), calcific deposits (calcific tendinitis), superior migration of humeral head (massive rotator cuff tear), AC joint OA, and fractures. MBS Item 58503.
MBS Available
Shoulder Ultrasound
First-line for rotator cuff pathology — high sensitivity and specificity for full-thickness tears; dynamic assessment; cost-effective; no radiation. Operator-dependent. MBS Item 55800 (with referral).
Specialist Referral
MRI Shoulder
Gold standard for soft-tissue assessment: partial-thickness tears, labral tears (SLAP), biceps pathology, chondral lesions. Recommended when surgery is considered or diagnosis remains unclear after ultrasound. MBS Item 63501 (with specialist referral).
Specialist
Diagnostic Subacromial Injection
Injection of local anaesthetic (± corticosteroid) into the subacromial bursa. Resolution of pain confirms subacromial pathology. Can be performed by GP or rheumatologist with ultrasound guidance (MBS Item 55801).

Special Populations

🤰 Pregnancy
Relaxin-induced ligamentous laxity
May predispose to GH instability and subluxation. Avoid MRI in first trimester unless essential. Ultrasound is preferred. Corticosteroid injections generally avoided in pregnancy.
De Quervain's tenosynovitis
Common in postpartum period; assess alongside shoulder examination. Wrist splinting is first-line.
👶 Paediatrics
Growth plate considerations
In adolescents, physeal injuries (Salter–Harris) mimic rotator cuff pathology. Clavicle fractures are the most common paediatric shoulder injury. Shoulder dislocation in patients <20 years has high recurrence rate (>90%).
Hypermobility
Generalised joint hypermobility (Beighton score) may cause multidirectional instability. Physiotherapy is first-line; surgical stabilisation reserved for recurrent dislocations.
👴 Elderly
Degenerative rotator cuff tears
Full-thickness rotator cuff tears are present in ~30% of people over 60, many asymptomatic. Pain and functional limitation — not imaging — should guide management.
PMR consideration
Bilateral shoulder girdle pain in patients >50 should prompt ESR/CRP testing for polymyalgia rheumatica. Responds rapidly to low-dose prednisolone (12.5–15 mg daily).
Fall risk
Proximal humerus fractures are the third most common fragility fracture after hip and distal radius fractures. Assess for osteoporosis risk. Consider DEXA scan (MBS Item 12312).
🫘 Renal Impairment
Dialysis-related amyloid
Beta-2-microglobulin amyloid deposits in long-term dialysis patients can cause shoulder pain, carpal tunnel syndrome, and destructive arthropathy. MRI shows periaricular amyloid deposits.
Calciphylaxis and metastatic calcification
CKD patients with secondary hyperparathyroidism may develop periarticular calcification mimicking calcific tendinitis.
🛡️ Immunocompromised
Septic arthritis
Immunocompromised patients (DMARD/biologic therapy, diabetes, HIV) have higher risk of septic arthritis. Acute shoulder pain + warmth + restricted active AND passive ROM warrants urgent aspiration and culture.
Opportunistic infection
Consider atypical organisms (mycobacteria, fungi) in immunosuppressed patients with chronic shoulder effusions unresponsive to standard therapy.

Aboriginal and Torres Strait Islander Health Considerations

Aboriginal and Torres Strait Islander Health
Epidemiology
Aboriginal and Torres Strait Islander peoples experience higher rates of musculoskeletal disease, including shoulder pathology, compared with non-Indigenous Australians. Musculoskeletal conditions are among the leading causes of disability in Indigenous Australians, with prevalence rates approximately 1.5–2 times higher than the general population (AIHW, 2023).
Remote access
Many Aboriginal and Torres Strait Islander peoples live in regional or remote areas with limited access to specialist rheumatology and orthopaedic services. Ultrasound and MRI availability is particularly limited in remote communities. Telehealth consultations can support clinical assessment, but physical examination remains essential.
Workforce & cultural safety
Engagement with Aboriginal Health Workers and Aboriginal Liaison Officers is important for culturally safe examination. Ensure informed consent, use of interpreters where needed, and respect for gender preferences in clinical examination. Involve family and community health workers in the examination process.
Occupational factors
Occupational shoulder injuries are common in labour-intensive industries disproportionately employing Indigenous Australians (agriculture, mining, construction). Repetitive overhead work and heavy manual labour contribute to rotator cuff pathology. Early assessment and workplace modification are key preventive strategies.
Diabetes & frozen shoulder
Type 2 diabetes is 3–4 times more prevalent in Aboriginal and Torres Strait Islander peoples compared with non-Indigenous Australians. Since diabetes is a major risk factor for frozen shoulder, the incidence of adhesive capsulitis is likely significantly higher. Diabetes screening should be considered in all Indigenous patients presenting with shoulder stiffness.
Management pathways
Primary care management in Aboriginal Community Controlled Health Organisations (ACCHOs) should include early physiotherapy referral, injection therapy where indicated, and supported referral pathways to regional or metropolitan specialist services. Patient-assisted travel schemes (PATS) are available in all Australian states for patients in remote areas requiring specialist review.

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